• 陕西省优秀期刊
  • 主办:陕西中医药大学

References:

[1]DU XM,CAI LY,XIE J,et al.The role of TGF-beta3 in cartilage development and osteoarthritis[J].Bone Research,2023,11(1):2.
[2]李强,庞仲辉,陶飞飞,等.萆薢总皂苷通过抑制软骨基质降解改善豚鼠骨关节炎的作用研究[J].现代中医药,2022,42(6):37-41.
[3]TATEBE M,IWATSUKI K,HIRATA H,et al.Effects of depression and inflammatory factors on chronic conditions of the wrist[J].The Bone & Joint Journal,2016,98-B(7):961-968.
[4]WU J,LI HL,HU F,et al.Stevioside attenuates osteoarthritis via regulating Nrf2/HO-1/NF-κB pathway[J].Journal of Orthopaedic Translation,2022,38:190-202.
[5]GóMEZ R,CONDE J,SCOTECE M,et al.What’s new in our understanding of the role of adipokines in rheumatic diseases?[J].Nature Reviews Rheumatology,2011,7(9):528-536.
[6]YAO N,PENG S,WU H,et al.Long noncoding RNA PVT1 promotes chondrocyte extracellular matrix degradation by acting as a sponge for miR-140 in IL-1β-stimulated chondrocytes[J].Journal of Orthopaedic Surgery and Research,2022,17(1):218.
[7]RAI MI,ALAM M,LIGHTFOOT DA,et al.Classification and experimental identification of plant long non-coding RNAs[J].Genomics,2019,111(5):997-1005.
[8]LIU YB,LIN LP,ZOU R,et al.MSC-derived exosomes promote proliferation and inhibit apoptosis of chondrocytes via lncRNA-KLF3-AS1/miR-206/GIT1 axis in osteoarthritis[J].Cell Cycle,2018,17(21/22):2411-2422.
[9]CROSS M,SMITH E,HOY D,et al.The global burden of hip and knee osteoarthritis:estimates from the global burden of disease 2010 study[J].Annals of the Rheumatic Diseases,2014,73(7):1323-1330.
[10]康朝宾,赵亚妮,卢延荣.温针灸联合灯盏细辛穴位注射治疗类风湿性关节炎临床研究[J].现代中医药,2022,42(6):117-121.
[11]FELLOWS CR,MATTA C,MOBASHERI A.Applying proteomics to study crosstalk at the cartilage-subchondral bone interface in osteoarthritis:current status and future directions[J].EBioMedicine,2016,11:2-4.
[12]LUI JC,GARRISON P,NGUYEN Q,et al.EZH1 and EZH2 promote skeletal growth by repressing inhibitors of chondrocyte proliferation and hypertrophy[J].Nature Communications,2016,7:13685.
[13]HE CL,GAO H,FAN XN,et al.Identification of a novel miRNA-target gene regulatory network in osteosarcoma by integrating transcriptome analysis[J].International Journal of Clinical and Experimental Pathology,2015,8(7):8348-8357.
[14]KNUDSON CB,KNUDSON W.Cartilage proteoglycans[J].Seminars in Cell & Developmental Biology,2001,12(2):69-78.
[15]CS-SZAB G,MELCHING LI,ROUGHLEY PJ,et al.Changes in messenger RNA and protein levels of proteoglycans and link protein in human osteoarthritic cartilage samples[J].Arthritis and Rheumatism,1997,40(6):1037-1045.
[16]LI HZ,XU XH,LIN N,et al.Overexpression of miR-10a-5p facilitates the progression of osteoarthritis[J].Aging,2020,12(7):5948-5976.
[17]CAI HQ,MIAO MY,WANG ZG.MiR-214-3p promotes the proliferation,migration and invasion of osteosarcoma cells by targeting CADM1[J].Oncology Letters,2018,16(2):2620-2628.
[18]SOPHIA FOX AJ,BEDI A,RODEO SA.The basic science of articular cartilage:structure,composition,and function[J].Sports Health,2009,1(6):461-468.
[19]SAITO T,FUKAI A,MABUCHI A,et al.Transcriptional regulation of endochondral ossification by HIF-2alpha during skeletal growth and osteoarthritis development[J].Nature Medicine,2010,16(6):678-686.
[20]AL-MODAWI RN,BRINCHMANN JE,KARLSEN TA.Multi-pathway protective effects of microRNAs on human chondrocytes in an in vitro model of osteoarthritis[J].Molecular Therapy Nucleic Acids,2019,17:776-790.
[21]WANG YJ,SHEN SP,LI Z,et al.MIR-140-5p affects chondrocyte proliferation,apoptosis,and inflammation by targeting HMGB1 in osteoarthritis[J].Inflammation Research,2020,69(1):63-73.
[22]LAFONT JE,TALMA S,HOPFGARTEN C,et al.Hypoxia promotes the differentiated human articular chondrocyte phenotype through SOX9-dependent and-independent pathways[J].The Journal of Biological Chemistry,2008,283(8):4778-4786.
[23]SHAO J,ZHANG Y,YANG TY,et al.HIF-1α disturbs osteoblasts and osteoclasts coupling in bone remodeling by up-regulating OPG expression[J].In Vitro Cellular & Developmental Biology-Animal,2015,51(8):808-814.
[24]XUE JF,SHI ZM,ZOU J,et al.Inhibition of PI3K/AKT/mTOR signaling pathway promotes autophagy of articular chondrocytes and attenuates inflammatory response in rats with osteoarthritis[J].Biomedecine & Pharmacotherapie,2017,89:1252-1261.
[25]LIU SY,CAO CX,ZHANG YJ,et al.PI3K/Akt inhibitor partly decreases TNF-α-induced activation of fibroblast-like synoviocytes in osteoarthritis[J].Journal of Orthopaedic Surgery and Research,2019,14(1):425.
[26]李曼,张露蓉.中药白花蛇舌草抗炎作用研究进展[J].辽宁中医药大学学报,2021,23(10):164-167.
[27]孙林林,乔利,田振华,等.姜黄化学成分及药理作用研究进展[J].山东中医药大学学报,2019,43(2):207-212.
[28]刘梅,郭小红,孙全,等.温郁金的化学成分和药理作用研究进展[J].现代药物与临床,2021,36(1):204-208.
[29]张建业,张卫华,韩俊,等.丹参酮ⅡA抑制软骨细胞炎症、软骨基质降解的机制及对大鼠骨关节炎的影响[J].解放军医药杂志,2021,33(3):10-16.
[30]张金锋,徐志龙,吴梦,等.丹参酮ⅡA通过抑制信号通路延缓膝骨关节炎大鼠软骨退变及抑制局部炎症的研究[J].中国药学杂志,2021,56(23):1918-1926.
[31]骞立刚,孙博,于泊,等.丹酚酸B减轻骨关节炎模型大鼠软骨组织损伤[J].基础医学与临床,2023,43(1):159-164.
[32]KUPTNIRATSAIKUL V,DAJPRATHAM P,TAECHAARPORNKUL W,et al.Efficacy and safety of Curcuma domestica extracts compared with ibuprofen in patients with knee osteoarthritis:a multicenter study[J].Clinical Interventions in Aging,2014,9:451-458.
[33]SUN YF,LIU W,ZHANG H,et al.Curcumin prevents osteoarthritis by inhibiting the activation of inflammasome NLRP3[J].Journal of Interferon & Cytokine Research,2017,37(10):449-455.
[34]李亚楠,倪娟,方禹舜,等.姜黄素通过调控脂多糖诱导的炎症抑制软骨细胞凋亡[J].中成药,2021,43(7):1904-1908.
[35]HAN GT,ZHANG YB,LI HH.The combination treatment of curcumin and probucol protects chondrocytes from TNF-α induced inflammation by enhancing autophagy and reducing apoptosis via the PI3K-akt-mTOR pathway[J].Oxidative Medicine and Cellular Longevity,2021,2021:5558066.

Memo

基金项目:山东省中医药科技发展计划项目(2019-0113);山东省中医药科技项目(2020Q016)

Last Update: 2024-07-15
Copyright @ 2020 Editorial Office of Modern Traditional Chinese Medicine
 Address: Middle Section of Century Avenue, Xianyang, Shaanxi Province. Post Code: 712046
Tel: 029-38185250 Fax: 029-38185250 E-mail: shxzhzs@163.com Powered by: www.editorhome.cn