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References:

[1]Bridges D,Moorhead GBG.14-3-3 proteins:a number of functions for a numbered protein[J].Science's STKE,2005,2005(2296):re10.
[2]Fu H,Subramanian RR,Masters SC.14-3-3 proteins:structure,function,and regulation[J].Annual Review of Pharmacology and Toxicology,2000,40:617-647.
[3]Marzinke MA,Mavencamp T,Duratinsky J,et al.14-3-3ε and NAV2 interact to regulate neurite outgrowth and axon elongation[J].Archives of Biochemistry and Biophysics,2013,540(1-2):94-100.
[4]Toyo-oka K,Wachi T,Hunt RF,et al.14-3-3ε and ζ regulate neurogenesis and differentiation of neuronal progenitor cells in the developing brain[J].The Journal of Neuroscience:the Official Journal of the Society for Neuroscience,2014,34(36):12168-12181.
[5]Berg D,Holzmann C,Riess O.14-3-3 proteins in the nervous system[J].Nature Reviews Neuroscience,2003,4(9):752-762.
[6]Zhang JJ,Zhou Y.14-3-3 proteins in glutamatergic synapses[J].Neural Plasticity,2018,2018:8407609.
[7]Pair FS,Yacoubian TA.14-3-3 proteins:novel pharmacological targets in neurodegenerative diseases[J].Trends in Pharmacological Sciences,2021,42(4):226-238.
[8]Mignon-Ravix C,Cacciagli P,El-Waly B,et al.Deletion of YWHAE in a patient with periventricular heterotopias and pronounced corpus callosum hypoplasia[J].J Med Genet.2010,47(2):132-136.
[9]Foote M,Zhou Y.14-3-3 proteins in neurological disorders[J].International Journal of Biochemistry and Molecular Biology,2012,3(2):152-164.
[10]Moore BW,Perez VJ.Specific acidic proteins of the nervous system[J].Physiological & Biochemical Aspects of Neverous Integration,1967:343-359.
[11]Martin H,Patel Y,Jones D,et al.Antibodies against the major brain isoforms of 14-3-3 protein.An antibody specific for the N-acetylated amino-Terminus of a protein[J].FEBS Letters,1993,331(3):296-303.
[12]Obsil T,Obsilova V.Structural basis of 14-3-3 protein functions[J].Seminars in Cell & Developmental Biology,2011,22(7):663-672.
[13]Boston PF,Jackson P,Thompson RJ.Human 14-3-3 protein:radioimmunoassay,tissue distribution,and cerebrospinal fluid levels in patients with neurological disorders[J].Journal of Neurochemistry,1982,38(5):1475-1482.
[14]Li Y,Wu YY,Zhou Y.Modulation of inactivation properties of CaV2.2 channels by 14-3-3 proteins[J].Neuron,2006,51(6):755-771.
[15]McFerrin MB,Chi XF,Cutter G,et al.Dysregulation of 14-3-3 proteins in neurodegenerative diseases with Lewy body or Alzheimer pathology[J].Annals of Clinical and Translational Neurology,2017,4(7):466-477.
[16]Antunes ASLM,Saia-Cereda VM,Crunfli F,et al.14-3-3proteins at the crossroads of neurodevelopment and schizophrenia[J].The World Journal of Biological Psychiatry,2021:1-19.
[17]Graham K,Zhang JJ,Qiao HF,et al.Region-specific inhibition of 14-3-3 proteins induces psychomotor behaviors in mice[J].NPJ Schizophrenia,2019,5(1):1.
[18]Lewis DA,Levitt P.Schizophrenia as a disorder of neurodevelopment[J].Annual Review of Neuroscience.2002,25(1):409-432.
[19]Sivagnanasundaram S,Crossett B,Dedova I,et al.Abnormal pathways in the genu of the corpus callosum in schizophrenia pathogenesis:a proteome study[J].Proteomics Clinical Applications,2007,1(10):1291-1305.
[20]English JA,Dicker P,Fcking M,et al.2-D DIGE analysis implicates cytoskeletal abnormalities in psychiatric disease[J].Proteomics,2009,9(12):3368-3382.
[21]English JA,Pennington K,Dunn MJ,et al.The neuroproteomics of schizophrenia[J].Biological Psychiatry,2011,69(2):163-172.
[22]Wong AHC,Likhodi O,Trakalo J,et al.Genetic and post-mortem mRNA analysis of the 14-3-3 genes that encode phosphoserine/threonine-binding regulatory proteins in schizophrenia and bipolar disorder[J].Schizophrenia Research,2005,78(2/3):137-146.
[23]Cheah PS,Ramshaw HS,Thomas PQ,et al.Neurodevelopmental and neuropsychiatric behaviour defects arise from 14-3-3ζ deficiency[J].Molecular Psychiatry,2012,17(4):451-466.
[24]Jaehne EJ,Ramshaw H,Xu XJ,et al.In-vivo administration of clozapine affects behaviour but does not reverse dendritic spine deficits in the 14-3-3ζ KO mouse model of schizophrenia-like disorders[J].Pharmacology,Biochemistry,and Behavior,2015,138:1-8.
[25]Xu XJ,Jaehne EJ,Greenberg Z,et al.14-3-3ζ deficient mice in the BALB/c background display behavioural and anatomical defects associated with neurodevelopmental disorders[J].Scientific Reports,2015,5:12434.
[26]Vawter MP ,Barrett T,Cheadle C,et al.Application of cDNA microarrays to examine gene expression differences in schizophrenia[J].Brain Research Bulletin,2001,55(5):641-650.
[27]Qiao HF,Foote M,Graham K,et al.14-3-3 proteins are required for hippocampal long-term potentiation and associative learning and memory[J].The Journal of Neuroscience,2014,34(14):4801-4808.
[28]Foote M,Qiao HF,Graham K,et al.Inhibition of 14-3-3 proteins leads to schizophrenia-related behavioral phenotypesand synaptic defects in mice[J].Biological Psychiatry,2015,78(6):386-395.
[29]Wachi T,Cornell B,Toyo-Oka K.Complete ablation of the 14-3-3epsilon protein results in multiple defects in neuropsychiatric behaviors[J].Behavioural Brain Research,2017,319:31-36.
[30]El-Mallakh RS,Elmaadawi AZ,Loganathan M,et al.Bipolar disorder:an update[J].Postgraduate Medicine,2010,122(4):24-31.
[31]Potash JB,Zandi PP,Willour VL,et al.Suggestive linkage to chromosomal regions 13q31 and 22q12 in families with psychotic bipolar disorder[J].The American Journal of Psychiatry,2003,160(4):680-686.
[32]Fallin MD,Lasseter VK,Avramopoulos D,et al.Bipolar I disorder and schizophrenia:a 440-single-nucleotide polymorphism screen of 64 candidate genes among AshkenaziJewish case-parent trios[J].American Journal of Human Genetics,2005,77(6):918-936.
[33]Higgs BW,Elashoff M,Richman S,et al.An online database for brain disease research[J].BMC Genomics,2006,7:70.
[34]Grover D,Verma R,Goes FS,et al.Family-based association of YWHAH in psychotic bipolar disorder[J].American Journal of Medical Genetics Part B,Neuropsychiatric Genetics:the Official Publication of the International Society of Psychiatric Genetics,2009,150B(7):977-983.
[35]Pers TH,Hansen NT,Lage K,et al.Meta-analysis of heterogeneous data sources for genome-scale identification of risk genes in complex phenotypes[J].Genetic Epidemiology,2011,35(5):318-332.
[36]Jacobsen KK,Kleppe R,Johansson S,et al.Epistatic and gene wide effects in YWHA andaromatic amino hydroxylase genes across ADHD and other common neuropsychiatric disorders:Association with YWHAE[J].American Journal of Medical Genetics Part B,Neuropsychiatric Genetics:the Official Publication of the International Society of Psychiatric Genetics,2015,168(6):423-432.
[37]Elashoff M,Higgs BW,Yolken RH,et al.Meta-analysis of 12 genomic studies in bipolar disorder[J].Journal of Molecular Neuroscience:MN,2007,31(3):221-243.
[38]Liu J,Li ZQ,Li JY,et al.Polymorphisms and haplotypes in the YWHAE gene increase susceptibility to bipolar disorder in Chinese Han population[J].The Journal of Clinical Psychiatry,2012,73(10):e1276-e1282.
[39]Mughal S, Faizy RM, Saadabadi A. Autism Spectrum Disorder[J].2020 Nov 18. In:StatPearls [Internet].Treasure Island (FL):StatPearls Publishing; 2021 Jan-. PMID:30247851.
[40]Bourgeron T.From the genetic architecture to synaptic plasticity in autism spectrum disorder[J].Nature Reviews Neuroscience,2015,16(9):551-563.
[41]Bruno DL,Anderlid BM,Lindstrand A,et al.Further molecular and clinical delineation of co-locating 17p13.3 microdeletions and microduplications that show distinctive phenotypes[J].Journal of Medical Genetics,2010,47(5):299-311.
[42]Pagan C,Goubran-Botros H,Delorme R,et al.Disruption of melatonin synthesis is associated with impaired 14-3-3 and miR-451 levels in patients with autism spectrum disorders[J].Scientific Reports,2017,7(1):2096.
[43]Rossignol DA,Frye RE.Melatonin in autism spectrum disorders[J].Current Clinical Pharmacology,2014,9(4):326-334.
[44]Scholz C,Steinemann D,Mlzer M,et al.NCAM2 deletion in a boy with macrocephaly and autism:Cause,association or predisposition?[J].European Journal of Medical Genetics,2016,59(10):493-498.
[45]Parcerisas A,Pujadas L,Ortega-Gascó A,et al.NCAM2 regulates dendritic and axonal differentiation through the cytoskeletal proteins MAP2 and 14-3-3[J].Cerebral Cortex (New York,N Y:1991),2020,30(6):3781-3799.
[46]Khandaker GM, Dantzer R. Is there a role for immune-to-brain communication in schizophrenia?[J].Psychopharmacology (Berl),2016 May,233(9):1559-1573.
[47]赵若莲, 王玉明, 段勇. 白细胞介素6与精神分裂症的研究进展[J].医学综述, 2009,(18):2757-2759.
[48]刘相辰, 苏雪丽, 赵丽珍. 药物联合电针治疗对慢性精神分裂症患者细胞免疫功能的影响[J].中国医药导报,2013,(6):12-14.

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基金项目:陕西中医药大学经脉-脏腑相关研究创新团队资助项目(YL-09)

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